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Through experimental methods such as molecular docking, CESTA, and non-targeted metabolomics, we found that SAC can act on the Keap1/Nrf2/GPX4 signaling axis and regulate glutathione metabolism, effectively inhibiting ferroptosis and oxidative damage in colonic epithelial cells, thereby alleviating UC

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Obesity and Oxidative Stress Evidence elucidates that obese women are more likely to suffer from several gynecological conditions, such as heavy or irregular menstrual bleeding, endometriosis, infertility, uterine fibroids, and polycystic ovary syndrome [39,40,41,42,43]