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Glutathione, which is synthesized in the cytoplasm but can cross into mitochondria, serves as a crucial buffer against oxidative damage to respiratory chain complexes and mitochondrial DNA.[4] Studies in both humans and rodent models demonstrate that restoring intracellular glutathione through glycine and NAC supplementation improves mitochondrial respiration, enhances ATP production, and reduces the oxidative damage accumulated within these critical organelles.[4] This improvement in mitochondrial efficiency has cascading benefits: better cellular energy production, improved metabolic health, and enhanced capacity for cellular repair and regeneration—all hallmarks of healthy aging

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This makes it challenging to quantify the synergistic effects of these risk factors fully