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in deoxygenated erythrocytes, 4 mg/mL inhibited HbS sickling by about 95.6% and reversed it by approximately 79.4% (Adejumo et al., 2012)

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Specific diseases caused by ABCG5 and ABCG8 gene defects: Sitosterolemia In contrast to bile acid and phospholipid transporters discussed above, ABCG5 and ABCG8 gene mutations primarily disrupt sterol homeostasis